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The Urinary System

Questions:
All 100

OBJ 2751 — Components & Functions

5 questions

OBJ 2752 — Kidney Structure

8 questions

OBJ 2753 — Blood Flow

8 questions

OBJ 2754 — Nephron & Urine Formation

14 questions

OBJ 2755 — Filtration & GFR

10 questions

OBJ 2756 — Tubular Fluid Changes

12 questions

OBJ 2757 — Ureters, Bladder & Urethra

7 questions

OBJ 2758 — Micturition

5 questions

OBJ 2759 — Water & Electrolyte Distribution

7 questions

OBJ 2760 — Fluid & Electrolyte Regulation

7 questions

OBJ 2761 — Buffer Systems

7 questions

OBJ 2762 — Acid-Base Threats

5 questions

OBJ 2763 — Aging

3 questions

OBJ 2764 — System Integration

2 questions

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The Urinary System

Martini, Ober, Bartholomew — Essentials of Anatomy & Physiology (Pearson, 2013) · Chapter 18

SOMAPL1JOBJ 2750–276415 Lesson Steps
2750
Medical Vocabulary — The Urinary System
Define the medical vocabulary components related to the urinary system.

Word Roots & Meanings

Root / PrefixMeaningExample Term
calyxa cup of flowersminor calyx — cup-shaped drain receiving urine from a renal papilla
detrudereto push downdetrusor muscle — smooth muscle of the bladder wall that compresses the bladder
fenestraa windowfenestrated capillaries — glomerular capillaries with endothelial pores
glomusa ballglomerulus — ball-shaped capillary network in the renal corpuscle
gonionangletrigone — triangular area on the floor of the urinary bladder
juxtanearjuxtaglomerular complex — endocrine structure near the glomerulus
micturireto urinatemicturition — the process of urination
nephroskidneynephron — the functional unit of the kidney
papillaesmall, nipple-shaped projectionsrenal papillae — tips of renal pyramids that discharge urine
podonfootpodocyte — cell with foot-like processes covering glomerular capillaries
rectusstraightvasa recta — straight capillaries paralleling the nephron loop
renkidneyrenal artery — artery supplying the kidney
retro-behindretroperitoneal — behind the peritoneum (kidney position)
vasavesselsvasa recta — straight vessels in the renal medulla
2751
Components of the Urinary System and Their Functions
Communicate the components of the urinary system and their functions.

Four Components

ComponentFunction
Kidneys (paired)Produce urine — a fluid containing water, ions, and small soluble compounds. Perform all excretory functions.
Ureters (paired)Transport urine from the renal pelvis to the urinary bladder via peristalsis.
Urinary bladderMuscular sac for temporary storage of urine prior to elimination.
UrethraConducts urine from the bladder to the exterior. In males, also transports semen.

Three Primary Functions

1
Excretion — removal of organic waste products from body fluids.
2
Elimination — discharge of these waste products into the environment.
3
Homeostatic regulation — regulating the volume and solute concentration of blood plasma.

Additional Homeostatic Functions

  • Regulating blood volume and blood pressure by adjusting water lost in urine, and releasing erythropoietin and renin
  • Regulating plasma ion concentrations (sodium, potassium, chloride, calcium via calcitriol synthesis)
  • Stabilizing blood pH by controlling loss of hydrogen ions (H⁺) and bicarbonate ions (HCO₃⁻)
  • Conserving nutrients — preventing excretion of glucose and amino acids while excreting nitrogenous wastes (urea and uric acid)
Clinical — Kidney FailureIf both kidneys fail, death occurs within days. Organic wastes accumulate to toxic levels, blood volume and pressure become unregulated, ion concentrations reach lethal levels, and pH control is lost. Treatment: dialysis or kidney transplant.
2752
Structural Features of the Kidneys
Identify the structural features of the kidneys.

Position and External Features

The kidneys lie on either side of the vertebral column between the last thoracic and third lumbar vertebrae. The right kidney sits slightly lower than the left. Both are retroperitoneal — behind the peritoneum, between the dorsal body wall muscles and the peritoneal lining.

Position is maintained by: (1) overlying peritoneum, (2) contact with adjacent organs, (3) supporting connective tissues — fibrous capsule, cushion of adipose tissue, and dense fibrous outer layer with collagen fibers. Damage to these supports = floating kidney — dangerous because ureters or blood vessels may twist.

A typical kidney is ~10 cm long, ~5.5 cm wide, ~3 cm thick, ~150 g. The hilum is the medial indentation where the ureter exits, and the renal artery/nerve enter and renal vein exits.

Internal Anatomy

Renal Cortex (outer)

Covers the medulla. Contains the renal corpuscles and convoluted tubules of nephrons. Renal columns extend inward between pyramids.

Renal Medulla (inner)

Contains 6–18 conical renal pyramids. Tips = renal papillae projecting into the renal sinus.

Urine Drainage Pathway (within kidney)

1
Urine produced in nephrons drains through renal papillae into minor calyces (cup-shaped drains).
2
4–5 minor calyces merge to form 2–3 major calyces.
3
Major calyces combine into the renal pelvis (large funnel-shaped chamber).
4
Renal pelvis connects to the ureter for drainage out of the kidney.

A renal lobe = one renal pyramid + overlying cortex + adjacent renal column tissue.

Each kidney has roughly 1.25 million nephrons with a combined tubular length of ~145 km (85 miles).

2753
Path of Blood Flow Through a Kidney
Identify the path of blood flow through a kidney.

Blood Supply Overview

The kidneys receive 20–25% of total cardiac output — about 1200 mL/min for organs weighing less than 300 g combined.

Arterial Pathway

1
Renal artery (from abdominal aorta) enters at the hilum.
2
Interlobar arteries — radiate outward between renal pyramids.
3
Arcuate arteries — arch along the cortex-medulla boundary.
4
Cortical radiate arteries (interlobular arteries) — supply the cortex.
5
Afferent arterioles — deliver blood to individual nephrons.
6
Glomerulus (capillary network inside renal corpuscle).
7
Efferent arteriole — blood exits the glomerulus.
8
Peritubular capillaries — surround PCT and DCT; pick up reabsorbed substances.

Venous Pathway (mirrors arterial)

Peritubular capillaries/vasa recta → cortical radiate veins → arcuate veins → interlobar veins → renal vein → inferior vena cava.

Two Types of Nephrons

Cortical Nephrons (~85%)

Located mostly within the cortex. Shorter nephron loops. Peritubular capillaries only — no vasa recta.

Juxtamedullary Nephrons (~15%)

Located near the medulla. Long nephron loops extending deep into medulla. Peritubular capillaries connect to vasa recta (long, straight capillaries paralleling the loop). Enable production of concentrated urine.

Unique FeatureThe nephron has an arteriole-to-arteriole arrangement (afferent → glomerulus → efferent). This is unique in the body and allows precise control of filtration pressure.
2754
Structure of the Nephron and Urine Formation
Identify the structure of the nephron and the processes involved in urine formation.

Nephron = Functional Unit of the Kidney

Each nephron has two main parts: (1) a renal corpuscle and (2) a 50-mm-long renal tubule.

The Renal Corpuscle

Consists of the glomerular (Bowman's) capsule surrounding the glomerulus (capillary network). Blood arrives via the afferent arteriole and departs via the efferent arteriole. Blood pressure forces fluid and dissolved solutes out of glomerular capillaries into the capsular space — this is filtration. The resulting protein-free solution is called filtrate.

The Filtration Membrane

Three layers that determine what passes into the capsular space:

1
Fenestrated endothelium of glomerular capillaries — endothelial cells contain pores.
2
Basement membrane — thick layer between endothelial cells and podocytes.
3
Filtration slits — narrow gaps between pedicels (foot processes) of podocytes covering the capillaries.

The filtration membrane blocks blood cells and most plasma proteins but permits water, metabolic wastes, ions, glucose, fatty acids, amino acids, and vitamins.

Segments of the Renal Tubule

SegmentLocationPrimary Function
Proximal convoluted tubule (PCT)CortexReabsorbs 60–70% of filtrate: all organic nutrients (glucose, amino acids), plasma proteins, and ions (Na⁺, K⁺, Ca²⁺, Mg²⁺, HCO₃⁻, etc.). Some H⁺ secretion.
Nephron loop (loop of Henle)Extends into medullaDescending limb: permeable to water, not solutes — water exits by osmosis. Ascending limb: impermeable to water AND solutes — actively pumps Na⁺ and Cl⁻ out. Creates medullary concentration gradient.
Distal convoluted tubule (DCT)CortexActive secretion of ions, acids, drugs, toxins. Selective reabsorption of Na⁺ (aldosterone-controlled) and water (ADH-controlled).

The Collecting System

DCT empties into collecting ductspapillary ducts → minor calyx. The collecting system makes final adjustments: reabsorbing water, and reabsorbing or secreting sodium, potassium, hydrogen, and bicarbonate ions.

The Juxtaglomerular Complex

Located where the DCT contacts the afferent arteriole. Consists of: (1) macula densa — tall epithelial cells at DCT start that monitor tubular fluid, and (2) juxtaglomerular cells — smooth muscle cells in the afferent arteriole wall. Secretes renin (enzyme for blood pressure regulation) and erythropoietin (hormone stimulating RBC production).

Three Nephron Processes

1
Filtration — blood pressure forces water and small solutes across the filtration membrane at the renal corpuscle. Passive process (no energy needed).
2
Reabsorption — removal of water and solutes from tubular fluid back into peritubular fluid and blood. Selective (uses carrier proteins). Water follows by osmosis.
3
Secretion — transport of solutes from peritubular fluid INTO tubular fluid. Catches substances missed by filtration, including drugs.
⚠ Key DistinctionThe PCT ignores urea and uric acid. As water and nutrients are reabsorbed, waste product concentrations rise progressively in the tubular fluid. This is how the kidney concentrates wastes while reclaiming useful substances.
Clinical — ProteinuriaIf the filtration membrane is damaged (e.g., podocyte disease), plasma proteins escape into the filtrate and appear in urine. Proteinuria is a hallmark of glomerular disease.
2755
Filtration Pressure and the Glomerular Filtration Rate
Identify the factors that influence filtration pressure and the rate of filtrate formation.

Filtration Pressure

Filtration pressure is the net force promoting filtration at the glomerulus. It is very low — approximately 10 mm Hg. Filtration pressure is higher than in other capillary beds because the efferent arteriole is slightly smaller in diameter than the afferent arteriole. This creates back-pressure that elevates glomerular capillary blood pressure.

Glomerular Filtration Rate (GFR)

GFR = the amount of filtrate produced per minute, averaging 125 mL/min. This means ~20% of blood delivered to the kidneys enters the capsular spaces. Daily filtrate production: ~180 liters (48 gallons) — 70 times total plasma volume. Over 99% is reabsorbed; only 700–2000 mL exits as urine.

Three Levels of GFR Regulation

LevelMechanismDetails
Autoregulation (local)Automatic diameter changes in afferent/efferent arteriolesLow blood flow: dilate afferent + constrict efferent → maintains glomerular pressure. High BP: constrict afferent → reduces flow.
HormonalRenin-angiotensin system, ADH, aldosterone, ANPLong-term adjustments to blood pressure and volume. Renin released when glomerular pressure is low.
Autonomic (sympathetic)Sympathetic vasoconstriction of afferent arteriolesDecreases GFR and shifts blood away from kidneys. During crisis (hemorrhage, heart attack), overrides autoregulation.

The Renin-Angiotensin System

Activated when glomerular pressures remain low (decreased blood volume, falling systemic pressure, renal artery blockage):

1
Juxtaglomerular complex releases renin into the circulation.
2
Renin converts angiotensinogen → angiotensin I.
3
ACE (in lung capillaries) converts angiotensin I → angiotensin II.
4
Angiotensin II: (a) vasoconstriction → raises BP, (b) constricts efferent arteriole → raises glomerular pressure, (c) triggers ADH release (water retention + thirst), (d) stimulates aldosterone secretion (Na⁺ retention).

ANP (atrial natriuretic peptide) opposes the renin-angiotensin system: released when blood volume/pressure is too high. Decreases sodium reabsorption, dilates glomerular capillaries, inhibits renin/aldosterone/ADH → more sodium and water lost in urine → lower blood volume and pressure.

Clinical — Exercise and Kidney FunctionDuring maximal exercise, renal blood flow may drop to less than 25% of resting levels. Metabolic wastes accumulate, glomerular cells may be damaged by hypoxia (causing protein or blood in urine), and in rare cases kidney failure occurs. Problems usually resolve within 48 hours.
2756
Tubular Fluid Changes from Filtrate to Urine
Identify the changes that occur in the tubular fluid as it moves through the nephron and exits as urine.

Three Metabolic Waste Products Excreted in Urine

WasteSourceDaily Production
Urea (most abundant)Amino acid breakdown~21 g/day
CreatinineCreatine phosphate breakdown in skeletal muscle~1.8 g/day
Uric acidRNA recycling/breakdown~480 mg/day

What Happens at Each Segment

SegmentFiltrate StatusKey Events
Renal corpuscleProtein-free plasmaFiltration produces filtrate. Same composition as plasma minus proteins.
PCT60–70% of volume reabsorbedAll glucose, amino acids, plasma proteins reabsorbed. Na⁺, K⁺, Ca²⁺, and other ions reabsorbed. Water follows by osmosis. Urea IGNORED — concentration rises.
Descending limbWater continues leavingPermeable to water, not solutes. Water exits by osmosis into hypertonic medullary interstitium. Volume drops further.
Ascending limbNa⁺/Cl⁻ removedImpermeable to water AND solutes. Actively pumps Na⁺ and Cl⁻ out. Tubular fluid becomes dilute (~1/3 plasma concentration). Creates medullary concentration gradient.
DCT~80% water, ~85% solutes goneActive secretion of ions, acids, drugs, toxins. Na⁺ reabsorbed (aldosterone). Water reabsorbed (ADH). K⁺/H⁺ secreted in exchange for Na⁺.
Collecting ductFinal adjustmentsADH determines water permeability: high ADH = concentrated urine; no ADH = dilute urine. Aldosterone adjusts Na⁺/K⁺ balance.

ADH and Urine Concentration

No ADH

DCT and collecting duct are impermeable to water. Large volume of dilute urine produced. Water cannot be reclaimed.

High ADH

DCT and collecting duct become highly permeable to water. Water reabsorbed by osmosis into concentrated medullary interstitium. Small volume of concentrated urine (up to 4–5x body fluid concentration).

Clinical — Diabetes InsipidusCaused by inadequate ADH secretion. DCT and collecting duct remain impermeable to water. Patient produces massive volumes of dilute urine → risk of fatal dehydration without treatment.
⚠ Why Urea ConcentratesThe PCT and nephron loop reabsorb water and useful solutes but largely ignore urea. As water volume decreases, the same amount of urea is dissolved in less fluid → urea concentration rises progressively. This is NOT active secretion of urea.

Normal Urine Properties

CharacteristicNormal Range
pH4.5–8 (average 6.0)
Specific gravity1.003–1.030
Osmolarity855–1335 mOsm/L
Volume700–2000 mL/day
Water content93–97%
Bacterial contentNone (sterile)
2757
Ureters, Urinary Bladder, and Urethra
Identify the structures and functions of the ureters, urinary bladder, and urethra.

The Ureters

Paired muscular tubes, ~30 cm (12 in.) long. Begin at the renal pelvis, end at the posterior wall of the urinary bladder without entering the peritoneal cavity. Ureteral openings in the bladder are slit-like (not round) — prevents backflow when bladder contracts.

Wall layers: inner transitional epithelium, middle longitudinal and circular smooth muscle, outer connective tissue. Peristaltic contractions every ~30 seconds sweep urine toward the bladder.

Clinical — Kidney Stones (Calculi)Solid deposits of calcium, magnesium salts, or uric acid crystals. Can form in the kidney, ureters, or bladder. Obstruct urine flow and may reduce or prevent filtration in the affected kidney. Cause severe pain (nephrolithiasis).

The Urinary Bladder

Hollow, muscular organ in the pelvic cavity. Only the superior surface is covered by peritoneum. Held in position by umbilical ligaments and connective tissue bands. Can contain up to ~1 liter of urine.

Males: between rectum and pubic symphysis. Females: inferior to uterus, anterior to vagina.

FeatureDescription
TrigoneTriangular area bounded by two ureteral openings and the urethral entrance. Urethral entrance at the apex (lowest point).
NeckArea surrounding the urethral entrance. Contains the internal urethral sphincter.
Detrusor muscleThree layers of smooth muscle (inner/outer longitudinal, middle circular). Contraction compresses bladder, expels urine into urethra.
Transitional epitheliumLines the bladder; continuous with renal pelvis and ureters. Can tolerate considerable stretching.

The Urethra

Extends from bladder neck to the exterior of the body.

Male Urethra

18–20 cm (7–8 in.). Extends to the external urethral orifice at the tip of the penis. Also transports semen.

Female Urethra

2.5–3.0 cm (~1 in.). Opens in the vestibule anterior to the vagina. Short length makes women more susceptible to UTIs.

Two Sphincters

SphincterMuscle TypeControl
Internal urethral sphincterSmooth muscleInvoluntary — provides automatic control over discharge
External urethral sphincterSkeletal muscleVoluntary — conscious control over urination
Clinical — Urinary Tract InfectionsCaused by bacteria or fungi colonizing the urinary tract (most commonly E. coli). Women especially susceptible due to short urethra near vagina and anus. Urethritis = inflamed urethra. Cystitis = inflamed bladder. Dysuria = painful urination. Can progress to pyelitis (renal pelvis) or pyelonephritis (cortex/medulla).
2758
The Process of Urination (Micturition)
Communicate the process of urination and how it is controlled.

The Micturition Reflex

1
As the bladder fills, stretch receptors in the bladder wall are stimulated.
2
Afferent sensory fibers in the pelvic nerves carry impulses to the sacral spinal cord.
3
Parasympathetic motor neurons bring close to threshold (local pathway) + interneurons relay sensation to thalamus → cerebral cortex (central pathway — conscious awareness).
4
Motor neurons stimulate the detrusor muscle to contract, elevating pressure inside the bladder.
5
Urine ejection requires relaxation of BOTH sphincters. The external sphincter relaxes under voluntary control; this then causes the internal sphincter to relax.

Key Volumes

  • Urge to urinate begins at ~200 mL
  • At >500 mL, the micturition reflex may force open the internal sphincter, followed by reflexive relaxation of the external sphincter — urination occurs despite voluntary opposition
  • After normal micturition, <10 mL remains in the bladder
Clinical — IncontinenceInability to control urination voluntarily. Causes: (1) Infants — corticospinal connections not yet established. (2) Stress incontinence — sphincter damage from childbirth; cough/sneeze overwhelms weakened sphincters. (3) Age-related — general loss of muscle tone. (4) Spinal cord injury — automatic bladder (reflex intact, voluntary control lost). (5) Pelvic nerve damage — eliminates reflex entirely; bladder distends, urine overflows uncontrolled.
2759
Water and Electrolyte Distribution in the Body
Identify how water and electrolytes are distributed within the body.

Body Water Content

Adult Males

~60% of total body weight is water (greater muscle mass — muscle is 75% water).

Adult Females

~50% of total body weight is water (relatively more adipose tissue — adipose is only 10% water).

Two Fluid Compartments

Intracellular Fluid (ICF) — ~2/3 of body water

Cytosol. Dominant ions: potassium (K⁺), magnesium, phosphate, and negatively charged proteins.

Extracellular Fluid (ECF) — ~1/3 of body water

Interstitial fluid + plasma + minor components (lymph, CSF, synovial fluid, serous fluids, aqueous humor). Dominant ions: sodium (Na⁺), chloride, bicarbonate.

Osmotic EquilibriumDespite major differences in specific ion concentrations, the ICF and ECF have identical osmotic concentrations. Osmosis eliminates any differences almost immediately because most plasma membranes are freely permeable to water. Changes in solute concentration → immediate water redistribution.

>90% of the osmotic concentration of the ECF results from sodium salts (primarily NaCl and NaHCO₃). Changes in ECF osmolarity almost always reflect changes in Na⁺ concentration.

2760
Fluid and Electrolyte Regulation
Communicate the basic concepts involved in the control of fluid and electrolyte regulation.

Three Interrelated Balances

  • Fluid balance — water gained daily = water lost daily. Maintained by creating ion concentration gradients that are eliminated by osmosis.
  • Electrolyte balance — no net gain or loss of any ion. Balances absorption (digestive tract) with excretion (kidneys).
  • Acid-base balance — H⁺ production = H⁺ loss. Maintaining body fluid pH within normal limits.

Daily Water Balance

Input (~2500 mL)Output (~2500 mL)
Water in food: ~1000 mL (40%)Urination: ~1200 mL
Liquid consumption: ~1200 mL (48%)Skin evaporation: ~750 mL
Metabolic water: ~300 mL (12%)Lung evaporation: ~400 mL
Feces: ~150 mL

Fluid Shifts

A fluid shift is water movement between the ECF and ICF in response to changes in osmotic concentration (osmolarity). Reaches equilibrium within minutes to hours.

  • ECF becomes hypertonic (more concentrated) → water moves from ICF into ECF (cells shrink)
  • ECF becomes hypotonic (more dilute) → water moves from ECF into cells (cells swell)

The ICF (larger volume) acts as a water reserve — prevents large changes in ECF osmolarity by distributing the change across both compartments.

Sodium Balance

Most common electrolyte balance problems involve sodium. Eating a heavily salted meal does NOT significantly raise Na⁺ concentration — osmosis brings water along from the digestive tract, diluting the sodium. But the ECF volume increases, which is why high-salt diets raise blood pressure.

Kidneys regulate Na⁺ loss: aldosterone stimulates Na⁺ reabsorption (decreases loss); ANP increases Na⁺ excretion.

Potassium Balance

~98% of body potassium is in the ICF. ECF K⁺ is normally low. Problems with K⁺ balance are less common but significantly more dangerous than Na⁺ imbalances (K⁺ directly affects cardiac function). When ECF K⁺ rises, aldosterone increases → K⁺ secreted into urine at the DCT. When ECF K⁺ falls, aldosterone decreases → K⁺ conserved.

2761
Buffering Systems for pH Balance
Communicate the buffering systems that balance the pH of the intracellular and extracellular fluids.

Three Major Buffer Systems

Buffer SystemPrimary LocationMechanism
Protein buffer systemsBoth ECF and ICFAmino acid side groups accept H⁺ when pH drops (amino group acts as weak base) or release H⁺ when pH rises (carboxyl group acts as weak acid). Plasma proteins and hemoglobin in RBCs are major contributors.
Carbonic acid–bicarbonate bufferPrimarily ECFCO₂ + H₂O ⇌ H₂CO₃ ⇌ H⁺ + HCO₃⁻. Carbonic acid (weak acid) and bicarbonate ion (weak base). Neutralizes metabolic acids. The bicarbonate reserve provides a large supply of HCO₃⁻ for this buffering.
Phosphate buffer systemPrimarily ICFH₂PO₄⁻ ⇌ H⁺ + HPO₄²⁻. Less important in ECF because HCO₃⁻ far exceeds phosphate concentration there. But phosphate concentration is high inside cells.

CO₂ and pH: The Inverse Relationship

When CO₂ levels RISE: more carbonic acid forms → more H⁺ released → pH FALLS (more acidic).

When CO₂ levels FALL: carbonic acid dissociates into CO₂ + H₂O → H⁺ removed from solution → pH RISES.

PCO₂ is the most important factor affecting pH in body tissues.

Respiratory Compensation

A change in respiratory rate that stabilizes ECF pH. Increasing breathing → more CO₂ exhaled → lower PCO₂ → pH rises. Decreasing breathing → CO₂ retained → higher PCO₂ → pH falls. Provides rapid pH adjustment.

Renal Compensation

Kidneys adjust rates of H⁺ secretion and HCO₃⁻ reabsorption. If pH too low (acidosis): secrete more H⁺, reabsorb more HCO₃⁻. If pH too high (alkalosis): secrete less H⁺, allow more HCO₃⁻ to be lost in urine. Slower than respiratory compensation but more thorough.

⚠ Buffers Are TemporaryBuffer systems tie up excess H⁺ but do not eliminate them. Once buffer molecules are used up, no more H⁺ can be absorbed and pH control becomes impossible. Respiratory and renal mechanisms must ultimately remove the captured H⁺ and regenerate buffer supplies.
2762
Threats to Acid-Base Balance
Identify the most frequent threats to acid-base balance.

Normal pH Range

ECF pH = 7.35–7.45. Below 7.35 = acidosis. Above 7.45 = alkalosis. Survival range: 6.8–7.7. Severe acidosis (pH < 7.0): CNS deterioration → coma, cardiac failure, circulatory collapse, death.

Four Acid-Base Disorders

DisorderpHCauseTreatment
Respiratory acidosis (MOST COMMON)<7.35Hypoventilation — CO₂ accumulates because it cannot be exhaled fast enough. Even minutes of hypoventilation can drop pH to 7.0.Improve ventilation (bronchodilation, mechanical assistance)
Metabolic acidosis (2nd most common)<7.35Production of excess metabolic acids (lactic acid, ketone bodies) OR impaired H⁺ excretion at the kidneys (severe kidney damage).Gradual bicarbonate administration + correct primary cause
Respiratory alkalosis (uncommon)>7.45Hyperventilation — excessive CO₂ loss (hypocapnia). Usually self-correcting as reduced PCO₂ removes chemoreceptor stimulation.Reduce respiratory rate, allow PCO₂ to rise
Metabolic alkalosis (rare in severe form)>7.45Prolonged vomiting — stomach generates replacement HCl, releasing HCO₃⁻ into blood each time. Bicarbonate accumulates, driving pH up.If pH >7.55: ammonium chloride administration
Acidosis Is More Common Than AlkalosisNormal metabolic operations generate acids (including carbonic acid). Several mechanisms exist to remove H⁺ (respiratory, renal, buffers), but the continuous acid production means the body is always defending against pH drops.
2763
Effects of Aging on the Urinary System
Identify the effects of aging on the urinary system.

Age-Related Changes

ChangeDetails
Decline in nephronsTotal number drops 30–40% between ages 25 and 85.
Reduced GFRFewer glomeruli, cumulative filtration damage, reduced renal blood flow. Also reduces ability to regulate pH through renal compensation.
Reduced ADH/aldosterone sensitivityDistal nephron and collecting system become less responsive. Result: reduced water and sodium reabsorption, increased potassium loss in urine.
Micturition problemsSphincter muscles lose tone → incontinence (slow leakage). CNS problems (stroke, Alzheimer's) → loss of voluntary control. Males: prostate enlargement compresses urethra → urinary retention.
Decreased total body waterAges 40–60: males 55%, females 47%. After 60: males ~50%, females ~45%. Less dilution of wastes, toxins, and administered drugs.
Net mineral lossAfter age 60, as muscle and skeletal mass decrease. Can be partially prevented by exercise and increased dietary mineral intake.
Increased systemic disordersMore disorders affecting major systems, many with impact on fluid, electrolyte, and/or acid-base balance.
2764
Urinary System Interactions with Other Body Systems
Communicate how the urinary system interacts with other body systems to maintain homeostasis in body fluids.

The urinary system provides the same fundamental service to every other body system: excreting waste products and maintaining normal body fluid pH and ion composition. Without this, no organ system can function.

SystemWhat It Does for the Urinary SystemWhat the Urinary System Does for It
IntegumentarySweat glands eliminate water/solutes; epidermis prevents excess fluid loss; produces vitamin D₃ for calcitriolEliminates nitrogenous wastes; maintains fluid/electrolyte/acid-base balance of blood nourishing skin
SkeletalAxial skeleton protects kidneys/ureters; pelvis protects bladder and proximal urethraConserves calcium and phosphate for bone growth
MuscularSphincter muscles control urination; trunk muscles protect urinary organsRemoves protein metabolism wastes; regulates Ca²⁺ and phosphate concentrations
NervousAdjusts renal blood pressure; monitors bladder distension; controls micturition reflexEliminates nitrogenous wastes; maintains blood composition critical for neural function
EndocrineAldosterone and ADH adjust rates of fluid and electrolyte reabsorptionKidney cells release renin (BP drops) and erythropoietin (O₂ levels fall); calcitriol production
CardiovascularDelivers blood to glomerular capillaries for filtration; accepts reabsorbed fluids and solutesReleases renin for BP regulation; releases EPO for RBC production
LymphaticProvides adaptive (specific) immune defense against UTIsEliminates toxins and wastes; acid pH of urine provides innate defense against infection
RespiratoryAssists in pH regulation by eliminating CO₂Assists in CO₂ elimination; provides bicarbonate buffers for pH regulation
DigestiveAbsorbs water for excretion; absorbs ions for normal fluid concentrations; liver removes bilirubinExcretes digestive toxins; excretes bilirubin and nitrogenous wastes from liver; calcitriol aids Ca²⁺/phosphate absorption

The Excretory System

The urinary system is the major component of an anatomically diverse excretory system that also includes:

  • Integumentary system — water, electrolytes, and small amounts of urea in perspiration
  • Respiratory system — CO₂ elimination; small amounts of acetone and water vapor
  • Digestive system — liver excretes metabolic wastes in bile; variable water loss in feces
The Big PictureThe urinary system excretes wastes and maintains normal body fluid pH and ion composition for ALL other body systems. The kidneys are far more closely regulated than the excretory functions of the integumentary, respiratory, or digestive systems.